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ST. ANDREWS, Scotland  The cheering jarred Inbee Park from her sleep.The 
10-year-old went downstairs to find her father in front of the television 
in the middle of the night in Seoul as he watched Se 
Ri Pak become the first South Korean to win the U.S. Women's 
Open.Within a week, Park wrapped her hands around a golf club for 
the first time, not knowing that it one day would lead her 
to the brink of history."They were doing replays every day on TV, 
her hitting the shot out of the water with her socks off," 
Park said. "It was cool to see her white feet. I didn't 
know what was happening, but I thought it was really cool to 
be seen playing golf and being on TV. Everybody was talking about 
it. Golf looked really fun."Fifteen years later, everyone is talking about 
Inbee Park.A win this week in the Women's British Open   
at St. Andrews, of all places    would make the 
25-year-old Park the first golfer to win four majors in one season.Arnold 
Palmer created the modern Grand Slam, winning four professional majors in 
one year. Jack Nicklaus, Tiger Woods and Annika Sorenstam got halfway home 
before their pursuit of it ended. No one has ever had a 
better shot at it than Park, who has won three LPGA Tour 
majors this year.She is a heavy favorite when the Open begins Thursday, 
just as Woods was at St. Andrews when he won to complete 
the career Grand Slam in 2000. Park already has won six times 
this year    half of those wins at majors  
  and has 
 told FoxNews.com. (The stem 
cells) can repair tissue damage caused by chemo radiotherapy, so those patients 
will tolerate chemotherapy much better. It gives enough room for clinicians 
to use a high dose of chemotherapy to kill cancer  and 
the patient can survive.Through a series of in vitro experiments, Geng and 
his team analyzed cells in the GI tract, stumbling upon an important 
molecule called ROBO1. They found that ROBO1 was specifically expressed 
in intestinal stem cells  but not in any other cells in 
the body. Upon this discovery, the researchers added to the cells a 
protein called SLIT2, which binds to ROBO1.The result: stem cell regeneration.Basically, 
you add SLIT2, you have more intestinal stem cells, Geng explained. If 
you have more intestinal stem cells, you repair more tissue damage, just 
like in general cell replication. So the ability to repair damage is 
higher  its just the logical explanation.The researchers theorized that 
by increasing stem cells in the gut, the intestine and GI tract 
are better protected from the effects of chemotherapy, allowing cancer patients 
to ingest nutrients and perform critical functions without releasing intestinal 
toxins into the blood circulation.To test this idea, Geng experimented with 
hundreds of mice with late-stage, metastatic cancer. All of the mice received 
a lethal dose of chemotherapy, but only half were given SLIT2 or 
an analogous protein called R-SPONDIN1 to stimulate intestinal 
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